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<article article-type="research-article" dtd-version="1.3" xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xml:lang="ru"><front><journal-meta><journal-id journal-id-type="publisher-id">cardiovascular</journal-id><journal-title-group><journal-title xml:lang="ru">Кардиоваскулярная терапия и профилактика</journal-title><trans-title-group xml:lang="en"><trans-title>Cardiovascular Therapy and Prevention</trans-title></trans-title-group></journal-title-group><issn pub-type="ppub">1728-8800</issn><issn pub-type="epub">2619-0125</issn><publisher><publisher-name>«SILICEA-POLIGRAF» LLC</publisher-name></publisher></journal-meta><article-meta><article-id pub-id-type="doi">10.15829/1728-8800-2013-1-95-101</article-id><article-id custom-type="elpub" pub-id-type="custom">cardiovascular-127</article-id><article-categories><subj-group subj-group-type="heading"><subject>Research Article</subject></subj-group><subj-group subj-group-type="section-heading" xml:lang="ru"><subject>ОБЗОРЫ</subject></subj-group><subj-group subj-group-type="section-heading" xml:lang="en"><subject>REVIEWS</subject></subj-group></article-categories><title-group><article-title>ПЕРИПРОЦЕДУРНОЕ ПОВРЕЖДЕНИЕ МИОКАРДА</article-title><trans-title-group xml:lang="en"><trans-title>PERIPROCEDURAL MYOCARDIAL DAMAGE</trans-title></trans-title-group></title-group><contrib-group><contrib contrib-type="author" corresp="yes"><name-alternatives><name name-style="eastern" xml:lang="ru"><surname>Акинина</surname><given-names>С. А.</given-names></name><name name-style="western" xml:lang="en"><surname>Akinina</surname><given-names>S. A.</given-names></name></name-alternatives><bio xml:lang="ru"><p>заместитель главного врача по терапии</p><p>Тел./факс: (3467) 39–01–67, 8 (902) 819-93-79 </p></bio><email xlink:type="simple">nordcardiolog@mail.ru</email><xref ref-type="aff" rid="aff-1"/></contrib></contrib-group><aff-alternatives id="aff-1"><aff xml:lang="ru"><institution>Учреждение Ханты-Мансийского автономного округа — Югры, «Окружная клиническая больница», Ханты-Мансийск</institution></aff><aff xml:lang="en"><institution>Khanty-Mansiysk Autonomous District — Yugra, District Clinical Hospital, Khanty-Mansiysk</institution></aff></aff-alternatives><pub-date pub-type="collection"><year>2013</year></pub-date><pub-date pub-type="epub"><day>20</day><month>02</month><year>2013</year></pub-date><volume>12</volume><issue>1</issue><fpage>95</fpage><lpage>101</lpage><permissions><copyright-statement>Copyright &amp;#x00A9; Акинина С.А., 2013</copyright-statement><copyright-year>2013</copyright-year><copyright-holder xml:lang="ru">Акинина С.А.</copyright-holder><copyright-holder xml:lang="en">Akinina S.A.</copyright-holder><license xml:lang="ru" license-type="creative-commons-attribution" xlink:href="https://creativecommons.org/licenses/by/4.0/" xlink:type="simple"><license-p>Данная работа распространяется под лицензией Creative Commons Attribution 4.0.</license-p></license><license xml:lang="en" license-type="creative-commons-attribution" xlink:href="https://creativecommons.org/licenses/by/4.0/" xlink:type="simple"><license-p>This work is licensed under a Creative Commons Attribution 4.0 License.</license-p></license></permissions><self-uri xlink:href="https://cardiovascular.elpub.ru/jour/article/view/127">https://cardiovascular.elpub.ru/jour/article/view/127</self-uri><abstract><p>Реваскуляризация миокарда методом чрескожных коронарных вмешательств (ЧКВ) широко и эффективно используется для лечения ишемической болезни сердца с достижением немедленного успеха &gt;90%. В зависимости от критериев диагностики, от 5% до 30% этих пациентов могут иметь признаки перипроцедурного повреждения миокарда (ППМ) или инфаркта миокарда (ПИМ). Предикторы развития ППМ, механизмы его появления и особенности клинической картины имеют важное значение в предупреждении ППМ. В определении и диагностике перипроцедурного некроза миокарда и ПИМ и их влияния на исходы до настоящего времени нет единой точки зрения. Согласно новейшим исследованиям, анализируемым в этом обзоре, в связи с высокой чувствительностью современных пороговых значений тропонина, возникло предложение о пересмотре современных критериев ППМ. </p></abstract><trans-abstract xml:lang="en"><p>Percutaneous coronary intervention (PCI), as a method of myocardial revascularisation, is widely and effectively used for the treatment of coronary heart disease (CHD), with immediate success rates of &gt;90%. Depending on the diagnostic criteria, 5–30% of these patients could develop the signs of periprocedural myocardial damage (PMD) or periprocedural myocardial infarction (PMI). PMD predictors, mechanisms of PMD development, and its specific clinical features play an important role in the PMI prevention. At present, there is no universal agreement on the definition and diagnostics of periprocedural myocardial necrosis and PMI, or on their impact on the clinical outcomes. According to the results of the recent studies, which are presented in this review, the current criteria of PMI might need to be modified, due to the increasingly high sensitivity of the modern threshold levels of troponin. </p></trans-abstract><kwd-group xml:lang="ru"><kwd>чрескожное коронарное вмешательство</kwd><kwd>перипроцедурный некроз миокарда</kwd><kwd>перипроцедурный инфаркт миокарда</kwd></kwd-group><kwd-group xml:lang="en"><kwd>percutaneous coronary intervention</kwd><kwd>periprocedural myocardial necrosis</kwd><kwd>periprocedural myocardial infarction</kwd></kwd-group></article-meta></front><back><ref-list><title>References</title><ref id="cit1"><label>1</label><citation-alternatives><mixed-citation xml:lang="ru">Cavallini C, Savonitto S, Violini R, et al. Impact of the elevation of biochemical markers of myocardial damage on long-term mortality after percutaneous coronary intervention: results of the CK-MB and PCI study. 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