Fatty acid and carbohydrate metabolism in acute myocardial ischemia
Abstract
Fatty acid metabolism (FA) requires 60-70% of O2 coming to myocardium. In O2 deficit, FA and carbohydrate metabolism declines. Reversible and irreversible ischemic damage is caused by unoxydated metabolites’ storage. Medicament therapy-induced carbohydrate metabolism is perspective, due to its less O2 demand.
About the Authors
M. A. AripovRussian Federation
D. Kh. Kamardinov
Russian Federation
S. V. Madoyan
Russian Federation
References
1. В.Дж. Маршалл. Клиническая биохимия 2000; 50-1.
2. MacDonald JIS, Sprecher H. Phospholipid fatty acid remodeling in mammalian cells. Biochim Biophys acta 1991;1084: 105-19.
3. Opie L. The Heart. Physiology and metabolism. New York 1991; 29-31.
4. Schulz H. Beta-oxidation of fatty acids. Biochim Biophys acta 1991; 1081: 110-4.
5. Thompson GR. A Handbook of hyperlipidaemia 1989 (London); 78-9.
6. Wojtczak L, Schonfeld P. Effect of fatty acid on energy coupling process in mitochondria. Biochim Biophys acta 1993; 1183: 51-7.
7. Chierchia SL. Метаболизм миокарда при стенокардии. Медикография 1999;.21(2): 45-50.
Review
For citations:
Aripov M.A., Kamardinov D.Kh., Madoyan S.V. Fatty acid and carbohydrate metabolism in acute myocardial ischemia. Cardiovascular Therapy and Prevention. 2005;4(5):95-98. (In Russ.)